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Diabetes
Diabetes has risen by over 14 percent in the last two years.
The CDC estimates that 20.8 million Americans -- 7 percent of
the U.S. population -- have diabetes, up from 18.2 million in
2003. [i][iii]
Centers for Disease Control Is a lack of
magnesium related to type 2 Diabetes in Obese Children? Dr.
Huerta and colleagues say yes in their study titled Magnesium
deficiency is associated with insulin resistance in obese
children.[ii][iv] Insulin resistance occurs when the body does
not use insulin, a protein made by the pancreas, to turn glucose
into energy. Children who are obese (seriously overweight) are
more likely to have insulin resistance. This might be because
they have low magnesium levels in their blood. This study was
done to see if obese children get enough magnesium in their
diets and if a lack of magnesium can cause insulin resistance
and eventually type 2 diabetes. This is the first study linking
low magnesium levels to insulin resistance in obese children.
Researchers found that 55% of obese children did not get enough
magnesium from the foods they ate, compared with only 27% of
lean children. Obese children had much lower magnesium
levels in their blood than lean children. Children with lower
magnesium levels had a higher insulin resistance.
The results of the diet survey showed that obese children got
14.4% less magnesium from the foods they ate than lean children.
An important finding was that even though obese and lean
children ate about the same number of calories per day, obese
children ate more calories from fatty foods than lean children.
In addition to not eating enough foods that have a lot of
magnesium, obese children might also have problems using
magnesium from the foods they eat. Extra body fat can prevent
the body’s cells from using magnesium to break down
carbohydrates.
When it comes to diabetes there is enough information pointing
to magnesium deficiency and chemical poisoning converging on the
young but the medical authorities would rather throw more money
into diabetic agencies to do more research. It is just too
difficult for them to simply address magnesium deficiencies.
What would be of greater benefit to these kids, research or
nutritional action? The United States government is involved in
a huge cover up of medical and pharmaceutical wrong doings and
will just keep on letting things slide as hundreds of thousands
of kids each year get sick.
Average levels of bad cholesterol and blood fats called
triglycerides were higher in youngsters who had the pre-diabetic
condition, in the study done by Dr. Hillier. One of the
extremely important things about this fact is what Dr. Carolyn
Dean shares about statins, magnesium and heart disease. Most
diabetics are put on statins, and this is one study which
clearly states that magnesium acts as a statin.[iii][v] If these
diabetic children were using magnesium, it would also prevent
them using these pharmaceutical drugs besides decreasing their
risk of CVD. Magnesium is what they need not the medical
establishment conducting more studies and research.
Type 2 diabetes can be controlled or prevented through weight
loss and by eating a healthier diet. Getting magnesium into the
kids at medicinal doses though is not easily achieved. Spirulina
and other super green foods like wheat grass juice are high in
absorbable magnesium in the form of chlorophyll. IV and
intramuscular injections are uncomfortable and expensive. Oral
magnesium supplements are not easily absorbable and some are
very high in lead. If we could get these magnesium levels in
these young children up and prevent diabetes, and if we could
raise the levels in the young who are already diagnosed
diabetics we would be preventing heart disease, stroke and many
other complications that all of these diabetics would eventually
face. This we can easily do with magnesium chloride applied
transdermally.
Magnesium is a wonder drug touted by pharmaceutical companies in
the emergency room and a simple element, a mineral vital to life
and health. Dr. Carolyn Dean said in her book The Miracle of
Magnesium, “The more I have learned about magnesium, the more
convinced I am that doctors are missing a huge opportunity by
not making it one of their “drugs of choice.” Because of the
disaster happening with children all doctors need to become
acutely aware of the great service they can do to prevent and
treat diabetes with magnesium.
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[i] American Diabetes Association
http://www.diabetes.org
[ii] Diabetes Care 28:1175–1181, 2005.
[iii] Rosanoff, Andrea. Seelig, Mildred. Comparison of Mechanism
and Functional Effects of Magnesium and Statin Pharmaceuticals.
Department of Physiology and Pharmacology, State University of
New York, Downstate Medical Center, Brooklyn (M.S.) Since
Mg2+-ATP is the controlling factor for the rate-limiting enzyme
in the cholesterol biosynthesis sequence that is targeted by the
statin pharmaceutical drugs, comparison of the effects of Mg2+
on lipoproteins with those of the statin drugs is warranted.
Formation of cholesterol in blood, as well as of cholesterol
required in hormone synthesis, and membrane maintenance, is
achieved in a series of enzymatic reactions that convert HMG-CoA
to cholesterol. The rate-limiting reaction of this pathway is
the enzymatic conversion of HMG CoA to mevalonate via HMG CoA.
The statins and Mg inhibit that enzyme. Large trials have
consistently shown that statins, taken by subjects with high
LDL-cholesterol (LDL-C) values, lower its blood levels 35 to
65%. They also reduce the incidence of heart attacks, angina and
other nonfatal cardiac events, as well as cardiac, stroke, and
total mortality. These effects of statins derive less from their
lowering of LDL-C than from their reduction of mevalonate
formation which improves endothelial function, inhibits
proliferation and migration of vascular smooth muscle cells and
macrophages, promotes plaque stabilization and regression, and
reduces inflammation, Mg has effects that parallel those of
statins. For example, the enzyme that deactivates HMG-CoA
Reductase requires Mg, making Mg a Reductase controller rather
than inhibitor. Mg is also necessary for the activity of
lecithin cholesterol acyl transferase (LCAT), which lowers LDL-C
and triglyceride levels and raises HDL-C levels. Desaturase is
another Mg-dependent enzyme involved in lipid metabolism which
statins do not directly affect. Desaturase catalyzes the first
step in conversion of essential fatty acids (omega-3 linoleic
acid and omega-6 linolenic acid) into prostaglandins, important
in cardiovascular and overall health. Mg at optimal cellular
concentration is well accepted as a natural calcium channel
blocker. More recent work shows that Mg also acts as a statin.
See also:
http://www.imva.info/diabetes.shtml More on this subject is available in the book
Transdermal Magnesium Therapy.
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